By Gregory Makowski
ISBN-10: 0128001410
ISBN-13: 9780128001417
ISBN-10: 0128003030
ISBN-13: 9780128003039
Volume 65 within the the world over acclaimed Advances in medical Chemistry comprises chapters authored by way of international well known medical laboratory scientists, physicians and examine scientists. The serial provides the most recent and most modern applied sciences concerning the sphere of medical Chemistry and is the benchmark for novel analytical methods within the scientific laboratory.
- Expertise of foreign contributors
- Latest state-of-the-art technologies
- Comprehensive in scope
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Pharmacol. 158 (2009) 1763–1770. J. Forrest, D. J. , Torcetrapib-induced blood pressure elevation is independent of CETP inhibition and is accompanied by increased circulating levels of aldosterone, Br. J. Pharmacol. 154 (2008) 1465–1473. [100] M. Guerin, W. Le Goff, E. , Inhibition of CETP by torcetrapib attenuates the atherogenicity of postprandial TG-rich lipoproteins in type IIB hyperlipidemia, Arterioscler. Thromb. Vasc. Biol. 28 (2008) 148–154. [101] H. Okamoto, F. Yonemori, K. Wakitani, T.
CONCLUSION The novel in vitro assay on the lipid transfers to HDL may independently predict the presence of CVD, and prospective studies are needed to confirm its importance as a CVD risk factor. It is challenging that the lipid transfers are altered in metabolic, inflammatory, and infectious diseases and can be modifiable by exercise training, dietary maneuvers, and drug treatments. To expand the capabilities of the method and the mechanisms behind the results, the overall relationships with HDL metabolism, composition, and subfraction profile should be explored, as well with all possible factors involved in lipid transfers.
Callegari, H. L. Catapano, HDL3 induces cyclooxygenase-2 expression and prostacyclin release in human endothelial cells via a p38 MAPK/CREdependent pathway: effects on COX-2/PGI-synthase coupling, Arterioscler. Thromb. Vasc. Biol. 24 (2004) 871–877. [59] T. Kimura, K. Sato, E. , High-density lipoprotein stimulates endothelial cell migration and survival through sphingosine 1-phosphate and its receptors, Arterioscler. Thromb. Vasc. Biol. 23 (2003) 1283–1288. [60] K. Tamama, H. Tomura, K. , High-density lipoprotein inhibits migration of vascular smooth muscle cells through its sphingosine 1-phosphate component, Atherosclerosis 178 (2005) 19–23.
Advances in Clinical Chemistry by Gregory Makowski
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